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A study on genetic linkages between aberrant lipid metabolism and PD directly demonstrates that glucocerebrosidase and sphingomyelin phosphodiesterase 1 mutations enhance PD risk in humans, and that galactosylceramide beta hydrolase function loss increases α-synuclein aggregation and accumulation in experimental murine models
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10.1016/j.jep.2021.114462 [DOI] [PubMed] [Google Scholar] 112.Liu DM, Zhou CY, Meng XL, Wang P, Li W
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